ERcast: Clinical Perspectives Podcast Preview

Hippo ERcast July 2024

  • Jul 2024
  • 9 Chapters
  • 2 hr 59 min

Welcome to the July 2024 Edition of ERcast!

This month, DeLaney, Andy, and Drew continue the discussion on taking time off and explore potential solutions. Next up, we have Chris Hahn and DeLaney walking us through cases where patients may have an occluded coronary artery yet the ECG doesn’t show an actual STEMI then Chris Hicks and Drew talk all things REBOA, Geoff and Drew give a heat-related illnesses refresher, Arun shares a consistent approach to managing distal radius fractures and Reuben Strayer and Andy discuss how to avoid errors in PSA. In Lit Matters, Drew and Cam Berg cover three articles on kidney disfunction. Let’s dive in!

Faculty

  • Cameron Berg, MD

    Based in Minneapolis, MN, Dr. Berg focuses on simplifying complex patient care processes, such as chest pain, syncope, and heart failure treatment. Since 2020, he has also been navigating his own recovery from a TBI after a bicycle accident. When he isn't in the clinic, Cameron is usually busy keeping his three young children alive and happy.

  • Drew Kalnow, DO

    Dr. Drew Kalnow is an emergency medicine physician and educator based in Columbus, Ohio. He completed his emergency medicine training at OhioHealth Doctors Hospital Emergency Medicine Residency. Dr. Kalnow is passionate about advancing emergency medicine through high-quality education, with a particular focus on simulation, learning theory, and innovative teaching.

  • Geoffrey Comp, DO, FACEP

    Dr. Comp is an Associate Program Director for the Creighton University / Valleywise Health Emergency Medicine Residency Program in Phoenix. A clinician-educator at heart, Geoff spends his time mentoring the next generation of Emergency Medicine residents and advocating for better ways to teach and learn medicine. His professional world revolves around wilderness medicine, clinician wellness, and finding innovative ways to bridge the gap between theory and the bedside. When he isn’t in the ED or the classroom, you’ll likely find him combining his love for medicine with his passion for the outdoors, always looking for a new trail to explore or a new way to collaborate with fellow clinicians.

  • Andy Little, DO

    Dr. Andy Little is an emergency medicine physician and educator. He earned his medical degree from the Ohio University Heritage College of Osteopathic Medicine and completed his emergency medicine residency at OhioHealth Doctors Hospital Emergency Medicine Residency, where he served as Chief Resident. He has received multiple national awards, including recognition from the American Osteopathic Association, American College of Osteopathic Emergency Physicians, and Emergency Medicine Residents' Association.

  • Chris Hicks, MD

    Chris Chris Hicks is an emergency physician, trauma team leader, educator, and speaker with expertise in resuscitation, simulation, and psychological performance in healthcare. His work has focused on areas such as mental practice, stress inoculation training, and improving team performance in high-stakes clinical environments. He has contributed to the development of interprofessional and simulation-based medical education initiatives and has collaborated with healthcare organizations on the design of systems, spaces, and teams to support high-performance care delivery. Chris is also a longtime supporter of the FOAMed movement and is widely recognized for his engaging and practical approach to medical education. Outside of medicine, he enjoys running, cycling, boxing, music, and spending time with his family.

  • Matthew DeLaney, MD, FACEP, FAAEM

    Dr. Matthew DeLaney is an emergency medicine physician and educator based in Birmingham, Alabama. A native of Mobile, he earned his medical degree from the University of South Alabama and completed his emergency medicine residency at Maine Medical Center.Dr. DeLaney has experience in both community and academic emergency medicine and is known for his commitment to teaching and medical education. He lives in Birmingham with his wife, Erin, who is also a physician, and their two daughters.

  • Arun Sayal, MD
  • Chris Hahn MD
  • Reuben Strayer, MD

Chapters

July Intro: A Solution to the ED Physician Vacation Woes?

Emergency physicians often struggle to take real vacation despite working shift-based schedules. Burnout rises when time off is fragmented or spent catching up before and after the trip, and practical fixes may require rethinking compensation, staffing, and how personal days are built into the calendar. ED Vacation Time Solutions Burnout and real time off: Vacation only helps when it is truly protected; fragmented days off and work that spills into travel time undermine recovery and track with higher burnout. Shift work paradox: Emergency medicine shares the shift-work structure seen in other hospital specialties, yet paid time off is often less explicit or harder to use in practice. Budget neutral tradeoffs: One proposed fix is exchanging some compensation for scheduled personal days, a financially realistic idea with meaningful cultural and staffing downsides. Pre and post vacation load: A common failure mode is pseudo-vacation, where clinicians work extra shifts before leaving and after returning, erasing much of the restorative benefit. Shop level policy options: The real question is which scheduling models are both palatable and sustainable across a group, and the practical tradeoffs are worth hearing in the episode.

The Hidden Widowmaker

Occlusive myocardial infarction often hides outside formal STEMI criteria, especially in subtle LAD occlusion. The key bedside shift is from millimeter cutoffs to proportional ECG interpretation: a shrinking QRS with relatively larger ST elevation or T waves should raise concern for OMI, not benign early repolarization. Recognizing Subtle LAD OMI OMI versus STEMI framing: Occlusive myocardial infarction is the more useful lens than STEMI/NSTEMI because patients with OMI can have similar infarct size and mortality despite never meeting classic STEMI criteria. Proportional ECG interpretation: Ratios matter more than raw millimeters: ST elevation and T-wave size should be judged against the preceding QRS, especially when the QRS is small. Shrinking R wave pattern: As myocardium becomes ischemic, the QRS and R waves may shrink while the T wave appears disproportionately larger, a subtle but high-yield clue to anterior OMI. Four variable formula: A 4-variable ECG calculator helps separate subtle anterior OMI from early repolarization using V2 QRS amplitude, V3 ST elevation, V4 R-wave amplitude, and QTc. We walk through where it actually helps in the episode. High risk clinical context: A scary chest pain story, dynamic serial ECG changes, and bedside wall-motion abnormalities all push slight ST elevation toward ischemia rather than a benign variant. Cath lab communication value: The formula should support rather than replace gestalt, but a score suggesting OMI can strengthen the case for urgent cardiology discussion and cath lab activation. When the Formula Does Not Apply QRS distortion exclusions: Anything that distorts the QRS, including LVH or left bundle branch block, can invalidate the formula because the proportional ECG relationships stop being reliable. Morphology based exclusions: Convex or coved ST morphology, terminal QRS distortion, and pathologic anterior Q waves are red flags that fall outside the calculator's intended use. T wave inversion limits: T-wave inversions in V2 through V6 are exclusion findings, so these patients need clinician-level ECG interpretation rather than a formula output. Clinical gestalt override: A reassuring calculator result should not overrule concern for ischemia in the wrong patient; standard chest pain evaluation and troponin testing still matter.

Lit Matters 1: The Experts Weigh In: IV Contrast in Patients with Kidney Disease

Modern IV iodinated contrast is safer in kidney disease than many clinicians were taught, and the biggest error is often delaying a needed contrast-enhanced CT. The key distinction is contrast-associated AKI versus true contrast-induced AKI, with risk driven mainly by baseline eGFR rather than contrast fear alone. IV Contrast in Kidney Disease CA-AKI versus CI-AKI: AKI within 48 hours of contrast is contrast-associated AKI, not proof of causation; that distinction explains why older contrast-nephropathy risk estimates likely overstated harm. Risk driven by eGFR: Baseline kidney function is the main signal, with CA-AKI rising stepwise as eGFR falls and the clearest concern concentrated in patients below 30 mL/min/1.73 m2. Low versus iso-osmolality contrast: Current evidence shows no confirmed clinically meaningful kidney-safety advantage of iso-osmolality over low-osmolality IV iodinated contrast for CT. Prophylaxis with IV fluids: Preventive treatment, when indicated, is IV volume expansion rather than N-acetylcysteine, and the patients most worth targeting are those with severe CKD who are not volume overloaded. We get into the practical caveats in the episode. Contrast as relative contraindication: Stage 4-5 CKD without maintenance dialysis is not an automatic reason to withhold contrast; if the scan answers a life-threatening question, contrast remains a relative rather than absolute contraindication. Single kidney and dose reduction: A solitary kidney does not add risk beyond the patient’s overall eGFR, and there is no evidence that empirically lowering contrast dose improves renal safety.

REBOA in the Community: Could We, Should We?

REBOA is a temporizing hemorrhage-control strategy for unstable abdominopelvic trauma, positioned as an endovascular alternative to resuscitative thoracotomy. The real question is not what the device is, but whether community emergency departments can use it safely, and where early femoral arterial access changes transfer readiness. Community ED REBOA in Trauma Endovascular aortic occlusion: REBOA places a balloon in the aorta to control noncompressible torso hemorrhage and may cause less physiologic disturbance than aortic cross-clamping in selected traumatic shock. Right patient population: The target patient is the unstable blunt or penetrating trauma patient with massive abdominal or pelvic bleeding who remains in shock despite volume resuscitation or blood products. Zone selection basics: Zone I is the workhorse for abdominal hemorrhage or a positive FAST in instability, while Zone III is reserved for pelvic or groin bleeding to preserve more distal organ perfusion. Femoral access first: Ultrasound-guided common femoral arterial access is the key prerequisite, and an established femoral arterial line can be exchanged for a REBOA catheter. We get into the practical handoff value of that move in the episode. Evidence and limitations: Most REBOA data come from trauma centers, and current literature has not shown a clear statistically significant outcome advantage strong enough to settle community adoption. Systems implementation barrier: Community use is not standard recommended practice in the US; successful rollout would require a shared protocol with the receiving trauma center rather than a solo device purchase.

Too Hot to Handle: Heat Emergencies

Heat stroke is a clinical diagnosis defined by hyperthermia plus central nervous system dysfunction, not by a single thermometer cutoff alone. In emergency medicine, heat illness spans cramps and syncope through heat exhaustion and life-threatening exertional or non-exertional heat stroke, with rapid cooling and safe disposition doing most of the work. Recognition and Differential of Heat Illness Clinical diagnosis over temperature: Heat-related illness is diagnosed by presentation rather than a number alone; hyperthermia with altered mental status should keep heat stroke high on the differential even when other causes are possible. Broad secondary hyperthermia differential: Not all hot patients have environmental heat illness, so infection, drugs or toxins, endocrine disease, and neurologic catastrophe still need parallel consideration in the altered, hyperthermic patient. Spectrum of heat presentations: Heat cramps, syncope, edema, exhaustion, and heat stroke sit on a clinical spectrum, with CNS dysfunction marking the jump from dehydration-type illness to true heat stroke. Exertional versus classic forms: Non-exertional heat stroke often affects older patients during heat waves and carries higher mortality, while exertional heat stroke classically presents in younger sweaty patients with profound dehydration. Hidden parallel emergencies: Prolonged heat exposure may be the consequence rather than the cause, so falls, stroke, myocardial infarction, and infection can coexist and change the whole management plan. We get into those bedside distinctions in the episode. Cooling Strategies and Disposition Cooling is the treatment: Heat stroke management is fundamentally about active and passive cooling; antipyretics do not help because hyperthermia is not a hypothalamic set-point problem. Passive cooling basics: Shade, airflow, loosened clothing, reduced ground conduction, and hydration all improve the body's own heat exchange and should start immediately while the team mobilizes more aggressive measures. Cold water immersion advantage: Cool water immersion is the fastest noninvasive option because water conducts heat about 24 times better than air, making it a high-yield strategy for severe exertional heat stroke. Evaporative and adjunct methods: Evaporative cooling, chemical cold packs, commercial devices, and chilled IV fluids all help, while benzodiazepines can reduce shivering or agitation that interferes with cooling. We walk through the practical setup in the chapter. Complications and safe disposition: Heat stroke can declare delayed transaminitis, DIC, rhabdomyolysis, and renal failure, so disposition hinges on end-organ risk and whether the patient can actually reach a safe cool environment after discharge.

Lit Matters 2: IV Contrast and Long-Term Kidney Function

Modern IV contrast for emergency CT imaging is not associated with meaningful long-term kidney dysfunction in most patients. In a large JAMA Internal Medicine study of ED patients evaluated for pulmonary embolism, contrast exposure showed no signal for lower 6-month eGFR, dialysis, or death. IV Contrast and Kidney Outcomes Long-term renal signal: Six-month kidney function was the primary outcome, and IV contrast was not associated with a clinically meaningful drop in eGFR after emergency diagnostic imaging. Acute kidney injury risk: Among patients with repeat creatinine checked within 7 days, AKI still showed no association with contrasted CT, reinforcing the newer data against contrast nephrotoxicity. Hard outcome reassurance: Kidney replacement therapy was rare at 0.11%, and contrast exposure was not linked to later dialysis or transplant after the index ED visit. Mortality outcome neutrality: All-cause mortality at 6 months was also unchanged, suggesting the absence of a detectable downstream harm signal from contrasted CTPA in this cohort. Regression discontinuity design: The study used a D-dimer cutoff to emulate a treatment-assignment boundary, a quasi-experimental design that strengthens causal inference beyond standard observational comparisons. We walk through why that method matters in the episode.

Ortho Wrist Reductions (When is it Good Enough?)

Distal radius fracture reduction is not just about making the X-ray pretty; acceptable alignment depends on age, function, and especially the plane of malalignment. Even clearly operative wrist fractures still benefit from ED reduction because better length and alignment reduce pain, swelling, and nerve compression risk. Distal Radius Reduction Pearls Key history and exam: Mechanism matters: distinguish mechanical from medical falls, high- from low-energy impact, and examine adjacent joints, skin, tendons, and multiple tenderness points so secondary injuries and open fractures are not missed. Acceptable versus anatomic alignment: The plane of deformity is the key lens: malalignment in a joint’s motion plane may remodel in children, while rotational or radial-ulnar deformity generally will not, a distinction we unpack in the episode. Age and remodeling potential: Children under about 15 to 16 have meaningful remodeling capacity, with younger patients remodeling more and girls losing that margin earlier as growth plates fuse sooner. Why reduce operative fractures: Even when surgery is likely, ED reduction still matters because restoring length and alignment decreases pain and swelling and can lower the risk of nerve impingement while patients await follow-up. TRAMP reduction framework: A reliable distal radius reduction follows TRAMP: Traction, Reduction, Apply splint, Mold, and Post-reduction X-ray. We walk through the bedside sequence and where the common misses happen in the chapter. Post-reduction angulation goal: Most wrists start with about 10 to 15 degrees of normal palmar tilt, so a good reduction aims for minimal residual dorsal angulation rather than accepting a visibly dorsally tilted position. Follow-up and Instability Red Flags Routine follow-up timing: Pediatric distal radius fractures should reach clinic within 7 days because they get sticky fast, while adults should generally be seen within 7 to 10 days. Unstable fracture patterns: Comminution, severe displacement, obliquity, and Smith fractures are less stable after reduction because fragment geometry and soft-tissue pull make redisplacement more likely. Intra-articular involvement: A distal radius fracture that extends into the joint deserves extra concern because even 1 to 2 mm of step-off can translate into post-traumatic arthritis. When follow-up is urgent: Unstable patterns need surgeon review as quickly as possible, particularly when the fracture is comminuted, intra-articular, markedly displaced, oblique, or volarly angulated. Second reduction decisions: If alignment is imperfect but follow-up is easy to obtain, another attempt may not help; when access is limited, a smarter second attempt and image review with orthopedics can be worth it.

Errors in Procedural Sedation & Analgesia

Procedural sedation is its own high-risk ED procedure, and the complication that matters most is hypoventilation. Safe PSA hinges on preparation, early recognition of respiratory decline, and choosing short-acting agents that match the pace of emergency care. Procedural Sedation Safety Hypoventilation as core hazard: PSA failures are usually respiratory, not procedural; hypoventilation from upper-airway obstruction or central apnea can be easy to miss when attention shifts to the reduction or repair. Preparation before first drug: A checklist-based setup matters more than any sedative choice: airway equipment, suction, monitoring, and full intubation capability including a paralytic should be at the bedside. Risk stratification upfront: Pulmonary comorbidity, difficult-airway features, and overall anesthetic risk should decide whether ED sedation is appropriate or whether delay, anesthesia support, or another strategy is safer. Oxygenation during PSA: Preoxygenation and continuous oxygen delivery are standard, with capnography layered into nasal cannula use and higher-level support considered for the highest-risk patients. We get into the setup nuances in the episode. Single-provider backup plan: If a second physician is not available, PSA still needs a dedicated in-room monitor; an additional RN focused on ventilation can catch respiratory decline before the pulse oximeter does. Recognizing and Treating Hypoventilation Early respiratory clues: Capnography and bedside observation matter because pulse oximetry can stay falsely reassuring during rising CO2, especially when supplemental oxygen is already on board. First rescue maneuvers: The safest first response is to stop sedatives, realign the head and neck, elevate the head of bed when possible, and perform a firm jaw thrust to reopen the airway. Bag-mask ventilation caution: Bag-mask ventilation is a rescue skill, but in PSA it also raises regurgitation risk, so simple airway maneuvers and positioning should be optimized before squeezing the bag. Readiness to intubate: Any PSA team must be prepared to convert immediately to definitive airway management if ventilation cannot be restored quickly. That threshold is worth hearing in the chapter. Medication Choices for PSA Retiring classic combinations: Fentanyl plus midazolam remains common, but its slower onset and longer duration make delayed hypoventilation more likely after the procedure seems finished. Faster on faster off: Propofol and ketamine fit ED procedural sedation better because they have rapid onset and shorter clinical duration, giving the operator tighter control over sedation depth. Conscious sedation misnomer: Moderate or conscious sedation implies responsiveness to voice or light touch, but PSA often requires a deeper level tailored to the procedure rather than the label. Fasting before ED PSA: ACEP does not recommend delaying emergency department PSA for fasting status alone, a practice point that still surprises many clinicians.

Lit Matters 3: Does the Amount of Contrast Given Affect Kidney Injury Rates?

Acute kidney injury after iodinated contrast is partly a dose phenomenon, especially in acute coronary syndrome patients heading to PCI. In this trial, lowering contrast volume reduced post-procedure AKI, while reinforcing a more practical ED point: order the contrasted study that answers the question, and avoid excess contrast when you can. Contrast Volume and Kidney Injury Dose dependent AKI signal: Post-PCI kidney injury tracked with contrast burden, supporting the idea that contrast-associated AKI is not all-or-none but rises as exposure increases. Randomized ACS PCI trial: A 550-patient randomized trial in acute coronary syndromes, most with STEMI, tested automated contrast-volume reduction against usual manual injection practice. Primary AKI reduction: Using less contrast lowered AKI from 24.3% to 16% within 48 hours, a clinically meaningful drop in a population already vulnerable to renal hypoperfusion. Baseline kidney function matters: The benefit was concentrated in patients with eGFR below 60, while patients with better baseline renal function did not show a clear difference. How much contrast changed: The reduction strategy cut contrast use by about 41%, roughly 95 mL versus 160 mL, which helps anchor how procedure volume compares with a typical contrasted CT. We get into that comparison in the episode. Practical ED takeaway: Contrast can transiently worsen renal function, but the bigger error is skipping the right contrasted study; appropriateness first, then minimize avoidable volume when feasible.